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His journey started in Nigeria, a taunted teenager with large tumors on his face, driven into deep despair.

Eleven years later, Victor Chukwueke has undergone numerous surgeries and is a step closer to achieving his dream of becoming a doctor.

In a rare act, the United States Congress passed a private bill last week granting Chukwueke permanent residency after years of his living in Michigan on an expired visa. The bill is awaiting President Barack Obama's signature.

"The day Congress passed the bill was one of the happiest days of my life," said Chukwueke, who left Nigeria as a teen in 2001 to get treatment for the tumors.

Private bills -- which only apply to one person and mostly focus on immigration -- seldom pass.

His is the only private bill to pass in Congress in two years.

"I was overwhelmed with joy; it was nothing less than a miracle," the 26-year-old said. "Only in this country can so many miraculous and wonderful things happen to someone like me."

Before coming to the United States at age 15, Chukwueke lived in the southeastern Nigeria town of Ovim.

He suffers from neurofibromatosis, a genetic disorder that causes massive life-threatening tumors on his face.

Treated as an outcast because of his deformed face, he was depressed and humiliated, he said. His family abandoned him at an orphanage.

Nuns from the Daughters of Mary Mother of Mercy rescued him from the orphanage more than a decade ago and arranged for a Michigan doctor to perform surgery on him.

He says he considers himself lucky to have developed the tumors.

"Without them, I would not have met the nun, left Nigeria, arrived in the U.S. and had the miracle to attend medical school," he says.

He lives with the nuns in Oak Park, Michigan. They have cared for him since he came to the U.S., where he has undergone seven surgeries, including one that left him blind in the right eye.

Doctors performed Chukwueke's surgeries over a period of time, he says, which contributed to his expired visa.

Despite the obstacles, he remains committed to getting an education.

"My own personal struggles to receive treatment have motivated and encouraged me to pursue a medical career ... to alleviate the pain and suffering of others," he says.

He finished his GED -- the equivalent of a high school education -- while undergoing treatment and enrolled at a community college.

A benefactor later helped him attend Wayne State University, where he graduated with a bachelor's degree in biochemistry last year. He had a 3.82 GPA and gave the university's commencement speech.

"Should I call myself a victim, or should I press forward to my dreams?" he asked during the speech amid thunderous applause.

Soon after his graduation, the University of Toledo in Ohio admitted him to medical school. The only hurdle: The program requires him to have permanent residency status, also known as a green card.

Though he qualifies for the DREAM Act, which gives immigrants who came to the United States as minors temporary residency, the measure would not give him the permanent status mandated by the university, according to his attorney.

And so began Chukwueke's journey to get legalized, a quest that has seen strangers rally to his help.

His attorney Thomas K. Ragland took his case pro bono.

"Victor's story is remarkable," said Ragland, who is based in Washington D.C. "Here is this kid who comes from Nigeria, he was taunted and teased for his diseases, and he comes to this country and excels, despite so many surgeries. It is a testament of not letting anything get in the way."

Sen. Carl Levin, a Michigan Democrat, sponsored the bill, S. 285. The measure passed the Senate in the summer and the House last week

"Already, his example has enriched Michigan and our nation, but I know that his contributions to our country are only beginning," Levin said in a statement.

The number of illegal immigrants in the United States was estimated at 11.5 million last year.

If Obama signs the bill, the State Department will reduce by one the number of immigrant visas available to Nigerians.

That signature, Chukwueke says, will be his favorite holiday gift.
http://edition.cnn.com/2012/12/23/us/michigan-nigerian-student/index.html?c=&page=0

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With the much-awaited launch of the iphone5 in Japan, the honour of being among the first to get their hands on the iPhone 5 has gone a bit awkward as thieves in Japan broke into carrier stores and carted away over $100,000 worth of Apple’s latest smartphone.

The Wall Street Journal reports that both Softbank and KDDI’s au were the victims of burglaries just hours before the iPhone 5 was set to go on sale. Three separate incidents across Osaka resulted in a total of 191 phones being lifted. Police estimated the largest heist, which saw 116 units nabbed from Softbank, was worth an estimated 7.45 million yen ($95,000).

It’s not clear whether the incidents were connected, though proximity and timing makes it possible that all three burglaries were committed by the same perpetrators.

The high value of Apple devices makes them frequent targets of theft. Apple has had difficulty with smash-and-grab burglaries that take advantage of the glass doors in many of its retail stores. One recent high-profile incident involved a BMW SUV crashing through a storefront in Temecula, California.

Friday morning’s burglaries do slightly mar the launch of Apple’s new gadget, but expected record sales should more than make up for the unfortunate incidents. Apple pre-sold more than 2 million iPhone 5 units in the first 24 hours of availability, and the company’s own estimates suggest new orders won’t be shipped for 3-4 weeks.
In nine countries around the world, legitimate owners of the iPhone 5 will begin purchasing the iPhone 5 on Friday. The Next Web obtained two units for testing the new HD Voice feature on the Telstra network in Australia. Call quality was clearly better with the devices, though it could still stand for improvement.

Teardown experts’ iFixit also made their way out to Australia to get early access to the iPhone 5. After breaking down the handset, they found it to be substantially more repairable than previous versions of the iPhone.

http://www.channelstv.com/home/2012/09/21/iphone-5-over-100000-stolen-in-japan-before-launch/?utm_source=&utm_medium=twitter

The woman who lost all seven children to mitochondria; a rare genetic condition
Sharon Bernardi lost all seven of her children to a rare genetic disease. It has driven her to support medical research that would allow defective genetic material to be replaced by DNA from another woman.


Sharon Bernardi and her son Edward, who died last year aged 21
Every time Sharon got pregnant she would pray that this time it would be different.

She felt fine during pregnancy and the births went well, and then quickly something would start to go wrong.

Each of her first three children died within hours of birth and no-one knew why. “It took us a long time to get over the first one and then it happened again. I was bewildered,” says Sharon, from Sunderland. “I was in shock.”

After the third child died, doctors began to suspect that the deaths weren’t coincidental. But genetic investigation didn’t provide any definite answers.

At the same time, her mother revealed that she’d had three stillbirths before Sharon had been born. Further investigations by doctors revealed that members of Sharon’s extended family had lost another eight children between them.

“I didn’t know about my mum’s history,” says Sharon. “There was no need for me to know. I was my mother’s only child. And I think that in her era people didn’t really talk about things as they do now.”

It’s hard when you want to have a family, and you finally have a baby… and then somebody tells you that at any moment your child is going to die” Sharon Bernardi

Then along came Edward, Sharon’s fourth child. This time the doctors were more prepared. For his first 48 hours, Edward received drugs and blood transfusions to prevent the lactic acidosis (a kind of blood poisoning) that had killed his siblings. Five weeks later Sharon and her husband Neil were allowed to take Edward to their home in Sunderland for Christmas.

Edward lived. Although his health was often poor and Sharon had to care for him a lot of the time, he was a cheerful, active boy. At the age of four he started to have seizures. It was then that the doctors were finally able to diagnose Edward’s – and indeed Sharon’s – problem.

Having gone through the history of Sharon’s babies, doctors diagnosed Edward with Leigh’s disease, a disorder that affects the central nervous system. The disease is caused by a defect in the mother’s mitochondria, often referred to as the power plant of the cell.

“This is going to sound strange but I was relieved that, at last, I had an answer.”

Not that the news made life much easier for Sharon. Her doctors told her that Edward could enjoy long periods of remission but that his health could also go down quite quickly. And meanwhile there was always the risk of Edward dying in one of his seizures, which could last for days.

“It’s hard when you want to have a family, and you finally have a baby like Edward, and you think you’re finally getting somewhere with your hopes and your dreams, and then somebody tells you that at any moment your child is going to die.”

Sharon and Neil Bernardi were told that Edward was going to die before he was five. “Obviously, you either go down or you start fighting,” says Sharon. Edward and his mother were fighters. In the end Edward survived into adulthood, dying last year at the age of 21.

Sharon and Neil kept on trying for a healthy baby but without luck. Although three more children were born, none lived beyond the age of two. Each time one of their children died, they told themselves that “the death was a one-off”. After their last child had a heart attack and died in 2000 they stopped trying.

“People ask, what was different about Edward? How come he survived as a baby when he had all the problems that would later build up? I don’t know but Edward had some fight in him. He was fighting to survive all his life. I think that was in his personality.”

The death of all her children put strains on her marriage, and on the wider family. “It also affects the family, the grandparents, their hopes and dreams for their grandchildren.”

People have accused Sharon and Neil of being selfish for wanting what they cannot have – their own family of healthy children. “I don’t think I am selfish,” says Sharon. “I wanted my child to be healthy.”

“In the last year of his life Edward was in chronic pain. He had dystonic spasms caused by things going wrong in his brain. His muscles would go into spasm for up to six hours at a time. Drugs could not help him. Part of Edward’s body was beginning to fail.”

The suffering of Sharon’s children has convinced her of the need to pursue the kind of genetic therapies that would allow mitochondrial defects to be remedied.

“When you see somebody in pain you don’t want to see somebody else in pain. You don’t want to see a child who is born only to suffer and die before they’re two, or if they do survive to have devastating disabilities.”

“It’s not about being selfish. It’s not about wanting designer babies. It’s not about doing injustice to people with disabilities. It’s about trying to create a healthy baby. It’s about trying to give a child a future.”

Facts about Leigh’s disease: Mitochondria

-Caused by problems in mitochondria – tiny structures that are power stations in every cell

-Particularly affects brain and nervous system

-Usually begins in early childhood

-Early signs include poor sucking ability, loss of head control and loss of acquired motor skills or movement

Science is researching three-parent IVF to address mitochondria

Public consultation has begun into ethics of using three people to create one baby

Technique can be used to prevent mitochondrial diseases passed from mother to child

Baby would have genetic information from two parents and donor woman

About one in 200 children born with faulty mitochondria

 

http://www.channelstv.com/home/2012/09/21/the-woman-who-lost-all-seven-children-to-mitochondria-a-rare-genetic-condition/?utm_source=&utm_medium=twitter

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