Ckushy's Posts
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teddybiyasi: .....good ![]() |
Yh, enough chit-chat the lines shall talk. |
paul thor, focus on your phoenix shit... Cos i didnt see the PHOENIX ORDER at the bottom. Are you tired ![]() |
Tiyazman: Vickybash, if i were you, i wouldn't reply these fools! Especially that Ice of a kid! He's the most annoying and dumbest Edo indigene i've ever encountered! The dude can be such a toilet!ok |
vickybash: what an epic painment! am really enjoying the show..1st,it was ckushy..then another cynic emerged.lol...if u didnt google vicky bash,how then did u know of the pics in my profile? hahaha..jobless troll.and the other bigger fool supported the clueless bastard..hahaha.why wnt u support him? birds of a feather..blind bats.keep the vibes on..keep searching for "vicky bash" on net.lol..its clear they cnt diff btw real name and nickname.hahaha..so funny..am loving thisGuy, you are a FAGG!!! Until you prove to us you have two teeths! I'm DONE with you!! TeddyB' drop something na, Its been long you dropped. Jigsaw a.k.a malik agars offspring, how far ![]() |
Dreal1247: Whether HIV or AIDS, i have no business with them.you sure? ![]() |
Read up |
...Once integrated, the virus may become latent, allowing the virus and its host cell to avoid detection by the immune system.[63] Alternatively, the virus may be transcribed, producing new RNA genomes and viral proteins that are packaged and released from the cell as new virus particles that begin the replication cycle anew.[64] Two types of HIV have been characterized: HIV-1 and HIV-2. HIV-1 is the virus that was originally discovered (and initially referred to also as LAV or HTLV-III). It is more virulent, more infective,[65] and is the cause of the majority of HIV infections globally. The lower infectivity of HIV-2 as compared with HIV-1 implies that fewer people exposed to HIV-2 will be infected per exposure. Because of its relatively poor capacity for transmission, HIV-2 is largely confined to West Africa.[66] Pathophysiology Main article: Pathophysiology of HIV/AIDS Scanning electron micrograph of HIV-1, colored green, budding from a cultured lymphocyte. After the virus enters the body there is a period of rapid viral replication, leading to an abundance of virus in the peripheral blood. During primary infection, the level of HIV may reach several million virus particles per milliliter of blood.[67] This response is accompanied by a marked drop in the number of circulating CD4+ T cells. The acute viremia is almost invariably associated with activation of CD8+ T cells, which kill HIV-infected cells, and subsequently with antibody production, or seroconversion. The CD8+ T cell response is thought to be important in controlling virus levels, which peak and then decline, as the CD4+ T cell counts recover. A good CD8+ T cell response has been linked to slower disease progression and a better prognosis, though it does not eliminate the virus.[68] Ultimately, HIV causes AIDS by depleting CD4+ T cells. This weakens the immune system and allows opportunistic infections. T cells are essential to the immune response and without them, the body cannot fight infections or kill cancerous cells. The mechanism of CD4+ T cell depletion differs in the acute and chronic phases.[69] During the acute phase, HIV-induced cell lysis and killing of infected cells by cytotoxic T cells accounts for CD4+ T cell depletion, although apoptosis may also be a factor. During the chronic phase, the consequences of generalized immune activation coupled with the gradual loss of the ability of the immune system to generate new T cells appear to account for the slow decline in CD4+ T cell numbers.[70] Although the symptoms of immune deficiency characteristic of AIDS do not appear for years after a person is infected, the bulk of CD4+ T cell loss occurs during the first weeks of infection, especially in the intestinal mucosa, which harbors the majority of the lymphocytes found in the body.[71] The reason for the preferential loss of mucosal CD4+ T cells is that the majority of mucosal CD4+ T cells express the CCR5 protein which HIV uses as a co-receptor to gain access to the cells, whereas only a small fraction of CD4+ T cells in the bloodstream do so.[72] A specific genetic change that alters the CCR5 protein when present in both chromosomes very effectively prevents HIV-1 infection.[73] HIV seeks out and destroys CCR5 expressing CD4+ T cells during acute infection.[74] A vigorous immune response eventually controls the infection and initiates the clinically latent phase. CD4+ T cells in mucosal tissues remain particularly affected.[74] Continuous HIV replication causes a state of generalized immune activation persisting throughout the chronic phase.[75] Immune activation, which is reflected by the increased activation state of immune cells and release of pro-inflammatory cytokines, results from the activity of several HIV gene products and the immune response to ongoing HIV replication. It is also linked to the breakdown of the immune surveillance system of the gastrointestinal mucosal barrier caused by the depletion of mucosal CD4+ T cells during the acute phase of disease.. . . . |
...HIV from a blood transfusion is extremely low (less than one in half a million) where improved donor selection and HIV screening is performed;[2] for example, in the UK the risk is reported at one in five million.[49] In low income countries, only half of transfusions may be appropriately screened (as of 2008),[50] and it is estimated that up to 15% of HIV infections in these areas come from transfusion of infected blood and blood products, representing between 5% and 10% of global infections.[2][51] Unsafe medical injections play a significant role in HIV spread in sub- Saharan Africa. In 2007, between 12 and 17% of infections in this region were attributed to medical syringe use.[52] The World Health Organisation estimates the risk of transmission as a result of a medical injection in Africa at 1.2%.[52] Significant risks are also associated with invasive procedures, assisted delivery, and dental care in this area of the world.[52] People giving or receiving tattoos, piercings, and scarification are theoretically at risk of infection but no confirmed cases have been documented. [53] It is not possible for mosquitoes or other insects to transmit HIV.[54] Mother-to-child HIV can be transmitted from mother to child during pregnancy, during delivery, or through breast milk.[55][56] This is the third most common way in which HIV is transmitted globally.[2] In the absence of treatment, the risk of transmission before or during birth is around 20% and in those who also breastfeed 35%.[55] As of 2008, vertical transmission accounted for about 90% of cases of HIV in children.[55] With appropriate treatment the risk of mother-to-child infection can be reduced to about 1%.[55] Preventive treatment involves the mother taking antiretroviral during pregnancy and delivery, an elective caesarean section, avoiding breastfeeding, and administering antiretroviral drugs to the newborn.[57] Many of these measures are however not available in the developing world.[57] If blood contaminates food during pre- chewing it may pose a risk of transmission.[53] Virology Main article: HIV A diagram showing the structure of HIV virus HIV is the cause of the spectrum of disease known as HIV/AIDS. HIV is a retrovirus that primarily infects components of the human immune system such as CD4+ T cells, macrophages and dendritic cells. It directly and indirectly destroys CD4+ T cells.[58] HIV is a member of the genus Lentivirus, [59] part of the family Retroviridae.[60] Lentiviruses share many morphological and biological characteristics. Many species of mammals are infected by lentiviruses, which are characteristically responsible for long-duration illnesses with a long incubation period.[61] Lentiviruses are transmitted as single- stranded, positive- sense, enveloped RNA viruses. Upon entry into the target cell, the viral RNA genome is converted (reverse transcribed) into double-stranded DNA by a virally encoded reverse transcriptase that is transported along with the viral genome in the virus particle. The resulting viral DNA is then imported into the cell nucleus and integrated into the cellular DNA by a virally encoded integrase and host co-factors |
Transmission Average per act risk of getting HIV by exposure route to an infected source Exposure route Chance of infection Blood transfusion 90% [27] Childbirth (to child) 25% Needle-sharing injection drug use 0.67%[27] Percutaneous needle stick 0.30%[29] Receptive anal intercourse* 0.04–3.0%[30] Insertive anal intercourse* 0.03%[31] Receptive penile- vaginal intercourse* 0.05–0.30%[30][32] Insertive penile- vaginal intercourse* 0.01–0.38% [30][32] Receptive oral intercourse*§ 0–0.04% [30] Insertive oral intercourse*§ 0–0.005%[33] * assuming no condom use § source refers to oral intercourse performed on a man HIV is transmitted by three main routes: sexual contact, exposure to infected body fluids or tissues, and from mother to child during pregnancy, delivery, or breastfeeding (known as vertical transmission).[2] There is no risk of acquiring HIV if exposed to feces, nasal secretions, saliva, sputum, sweat, tears, urine, or vomit unless these are contaminated with blood.[29] It is possible to be co-infected by more than one strain of HIV—a condition known as HIV superinfection.[34] Sexual The most frequent mode of transmission of HIV is through sexual contact with an infected person.[2] The majority of all transmissions worldwide occur through heterosexual contacts (i.e. sexual contacts between people of the opposite sex);[2] however, the pattern of transmission varies significantly among countries. In the United States, as of 2009, most sexual transmission occurred in men who had sex with men,[2] with this population accounting for 64% of all new cases.[35] With regard to unprotected heterosexual contacts, estimates of the risk of HIV transmission per sexual act appear to be four to ten times higher in low-income countries than in high-income countries. [36] In low-income countries, the risk of female-to-male transmission is estimated as 0.38% per act, and of male-to-female transmission as 0.30% per act; the equivalent estimates for high-income countries are 0.04% per act for female-to- male transmission, and 0.08% per act for male-to-female transmission.[36] The risk of transmission from anal intercourse is especially high, estimated as 1.4–1.7% per act in both heterosexual and homosexual contacts.[36][37] While the risk of transmission from MouthAction is relatively low, it is still present.[38] The risk from receiving MouthAction has been described as "nearly nil";[39] however, a few cases have been reported.[40] The per-act risk is estimated at 0–0.04% for receptive oral intercourse.[41] In settings involving prostitution in low income countries, risk of female-to-male transmission has been estimated as 2.4% per act and male-to- female transmission as 0.05% per act.[36] Risk of transmission increases in the presence of many sexually transmitted infections[42] and genital ulcers.[36] Genital ulcers appear to increase the risk approximately fivefold.[36] Other sexually transmitted infections, such as gonorrhea, chlamydia, trichomoniasis, and bacterial vaginosis, are associated with somewhat smaller increases in risk of transmission. [41] The viral load of an infected person is an important risk factor in both sexual and mother-to-child transmission.[43] During the first 2.5 months of an HIV infection a person's infectiousness is twelve times higher due to this high viral load.[41] If the person is in the late stages of infection, rates of transmission are approximately eightfold greater.[36] Commercial sex workers (including those in pornography) have an increased rate of HIV.[44][45] Rough sex can be a factor associated with an increased risk of transmission.[46] Sexual assault is also believed to carry an increased risk of HIV transmission as condoms are rarely worn, physical trauma to the vagina or rectum is likely, and there may be a greater risk of concurrent sexually transmitted infections.[47] Body fluids CDC poster from 1989 highlighting the threat of AIDS associated with drug use The second most frequent mode of HIV transmission is via blood and blood products.[2] Blood-borne transmission can be through needle-sharing during intravenous drug use, needle stick injury, transfusion of contaminated blood or blood product, or medical injections with unsterilised equipment. The risk from sharing a needle during drug injection is between 0.63 and 2.4% per act, with an average of 0.8%.[48] The risk of acquiring... |
...Although most HIV-1 infected individuals have a detectable viral load and in the absence of treatment will eventually progress to AIDS, a small proportion (about 5%) retain high levels of CD4+ T cells (T helper cells) without antiretroviral therapy for more than 5 years.[14][19] These individuals are classified as HIV controllers or long-term nonprogressors (LTNP).[19] Another group is those who also maintain a low or undetectable viral load without anti-retroviral treatment who are known as "elite controllers" or "elite suppressors". They represent approximately 1 in 300 infected persons. [20] Acquired immunodeficiency syndrome Main symptoms of AIDS. Acquired immunodeficiency syndrome (AIDS) is defined in terms of either a CD4+ T cell count below 200 cells per µL or the occurrence of specific diseases in association with an HIV infection.[14] In the absence of specific treatment, around half of people infected with HIV develop AIDS within ten years.[14] The most common initial conditions that alert to the presence of AIDS are pneumocystis pneumonia (40%), cachexia in the form of HIV wasting syndrome (20%) and esophageal candidiasis.[14] Other common signs include recurring respiratory tract infections.[14] Opportunistic infections may be caused by bacteria, viruses, fungi and parasites that are normally controlled by the immune system.[21] Which infections occur partly depends on what organisms are common in the person's environment. [14] These infections may affect nearly every organ system.[22] People with AIDS have an increased risk of developing various viral induced cancers including Kaposi's sarcoma, Burkitt's lymphoma, primary central nervous system lymphoma, and cervical cancer. [15] Kaposi's sarcoma is the most common cancer occurring in 10 to 20% of people with HIV.[23] The second most common cancer is lymphoma which is the cause of death of nearly 16% of people with AIDS and is the initial sign of AIDS in 3 to 4%.[23] Both these cancers are associated with human herpesvirus 8.[23] Cervical cancer occurs more frequently in those with AIDS due to its association with human papillomavirus (HPV).[23] Additionally, people with AIDS frequently have systemic symptoms such as prolonged fevers, sweats (particularly at night), swollen lymph nodes, chills, weakness, and weight loss.[24] Diarrhea is another common symptom present in about 90% of people with AIDS.[25] They can also be affected by diverse psychiatric and neurological symptoms independent of opportunistic infections and cancers. |
Signs and symptoms Main article: Signs and symptoms of HIV/ AIDS There are three main stages of HIV infection: acute infection, clinical latency and AIDS.[10][11] Acute infection Main symptoms of acute HIV infection The initial period following the contraction of HIV is called acute HIV, primary HIV or acute retroviral syndrome. [10][12] Many individuals develop an influenza-like illness or a mononucleosis- like illness 2–4 weeks post exposure while others have no significant symptoms.[13][14] Symptoms occur in 40–90% of cases and most commonly include fever, large tender lymph nodes, throat inflammation, a rash, headache, and/or sores of the mouth and genitals. [12][14] The rash, which occurs in 20– 50% of cases, presents itself on the trunk and is maculopapular, classically.[15] Some people also develop opportunistic infections at this stage.[12] Gastrointestinal symptoms such as nausea, vomiting or diarrhea may occur, as may neurological symptoms of peripheral neuropathy or Guillain-Barre syndrome.[14] The duration of the symptoms varies, but is usually one or two weeks.[14] Due to their nonspecific character, these symptoms are not often recognized as signs of HIV infection. Even cases that do get seen by a family doctor or a hospital are often misdiagnosed as one of the many common infectious diseases with overlapping symptoms. Thus, it is recommended that HIV be considered in people presenting an unexplained fever who may have risk factors for the infection.[14] Clinical latency The initial symptoms are followed by a stage called clinical latency, asymptomatic HIV, or chronic HIV.[11] Without treatment, this second stage of the natural history of HIV infection can last from about three years[16] to over 20 years[17] (on average, about eight years).[18] While typically there are few or no symptoms at first, near the end of this stage many people experience fever, weight loss, gastrointestinal problems and muscle pains.[11] Between 50 and 70% of people also develop persistent generalized lymphadenopathy, characterized by unexplained, non-painful enlargement of more than one group of lymph nodes (other than in the groin) for over three to six months.[10] |
Human immunodeficiency virus infection / acquired immunodeficiency syndrome (HIV/AIDS) is a disease of the human immune system caused by infection with human immunodeficiency virus (HIV).[1] The term HIV/AIDS represents the entire range of disease caused by the HIV virus from early infection to late stage symptoms. During the initial infection, a person may experience a brief period of influenza-like illness. This is typically followed by a prolonged period without symptoms. As the illness progresses, it interferes more and more with the immune system, making the person much more likely to get infections, including opportunistic infections and tumors that do not usually affect people who have working immune systems. HIV is transmitted primarily via unprotected sexual intercourse (including anal and MouthAction), contaminated blood transfusions, hypodermic needles, and from mother to child during pregnancy, delivery, or breastfeeding.[2] Some bodily fluids, such as saliva and tears, do not transmit HIV.[3] Prevention of HIV infection, primarily through safe sex and needle-exchange programs, is a key strategy to control the spread of the disease. There is no cure or vaccine; however, antiretroviral treatment can slow the course of the disease and may lead to a near-normal life expectancy. While antiretroviral treatment reduces the risk of death and complications from the disease, these medications are expensive and have side effects. Without treatment, the average survival time after infection with HIV is estimated to be 9 to 11 years, depending on the HIV subtype.[4] Genetic research indicates that HIV originated in west-central Africa during the late nineteenth or early twentieth century.[5] AIDS was first recognized by the United States Centers for Disease Control and Prevention (CDC) in 1981 and its cause—HIV infection—was identified in the early part of the decade.[6] Since its discovery, AIDS has caused an estimated 36 million deaths worldwide (as of 2012). [7] As of 2012, approximately 35.3 million people are living with HIV globally.[7] HIV/ AIDS is considered a pandemic—a disease outbreak which is present over a large area and is actively spreading.[8] HIV/AIDS has had a great impact on society, both as an illness and as a source of discrimination. The disease also has significant economic impacts. There are many misconceptions about HIV/AIDS such as the belief that it can be transmitted by casual non-sexual contact. The disease has also become subject to many controversies involving religion. It has attracted international medical and political attention as well as large-scale funding since it was identified in the 1980s.[9] |
the dude ran I'mma catch you later |
vickybash: 1st he's acting out of stupidity..next,he will be asking for my number to prove my gender.why dont u come clean? u're crushing on me,aint u? lol..keep it up dear..the only possible attention u'll get from me is what am giving to u nowcrush on you?? 'bleeped jollie,cindy lucas, akira,tori black... ![]() |
vickybash: seek for attention? lol..epic painment! u're pained cuz i got the name and attention u've been craving since God knows when.what name did you get On a social network?.. Bytch! I told you I googled your name and you think I did?? I can never ask page and brin 'bout a fagg like you. Guy, stop hiding in a bytch.. Your the one seeking for attention, you just popped outta nowhere when a tourney 'bout to kick off , you think Iono you?? Dude just sdfu and close your teeths![]() |
vickybash: u think u know "S.HIT?"..unscramble THIS!.u missed that part? lol..read my signature again - there is more to me than meets the eyes!!Dude, An infant'll unscramble THIS ![]() |
vickybash: seeking for attention? come back on ur 18th birthday cuz its obvious u're still a kid.now,troll with ur matesshutup! Close your teeths. I dont troll, I troll people like you, creating fake monikers to seek for attention. Until you prove to us you gat a bottomless pit, you a fagg!! |
vickybash: u shouldnt quote or reply every post.any drug head will easily decipher the puns in my versean infant would decipher those lines. Wtf's teeths and you think you dope Rhyming stick with teeths, kid with sleep... Hey! Your wack!!! Teeths/t!ts??A Bunch of monks in a sexx tape, your pun's really bad/A mosquito gets more claps than vickybash/crush on you?? Aint a fagg, 'Felt more buttts than the seats in cinemas/'Bleeped taylor,katty,Miley,Ariane even Nicki minaj!/ Why the fake monicker,you a fagg? You want head like criminals??Bytch. Erryline you pop is useless. Your dad bout to barge in and you still watching a brunette in a p0rno,you masturbatin,'bout to spit in a durex; What you bout to face, you clueless,you dont know!!! You can wake nostradamus. |
the firmness..buh u? even as my clone,u cnt prove 'abreast' of vicky cuz ur booobs have fallen zigzag..like my nanny's 'teeths!! ..//// teeths ![]() |
ELYMAXiimus: yeah you're right! But even though she's a 'he' its no body's biz! Nothing here is real!sorry, pls don't reply. YOUR A FUCCTARD. |
ELYMAXiimus: looool! Dude, she aint no guy! I've seen her on fbSo, I can't open a female account and upload a female picture??... I googled vicky bash, she uploaded only 1 picture, you telling me that aint weird?? Or she has another facebook account?. |
Jigsaw, you blaq as fucc Darker than Malik Agar's Knee cap. NASA!!! We found one
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Simonagbons: seems like biochemistry ain't hot in Nigeriawatchu mean ![]() |
Amadaz: Evil spirits never diesYoruba Films addict ![]() |
vickybash: what the heck happened here? ok..i know it..i know it.i will be back in a momentGuy, how far? ![]() |
JigsawKillah: how can someone buy a 'under'? The-nigga-dumb-die! U still yet to hit me cos u deliver-puns-like the 'old lady' u are, with imbalanced-fist, so if we hear say u dey top, na from italian-league!second thought, Aint replying you again:p :b your verses crack my ribs, stop trolling you more of a clown. Kpokpodipo ko kediki ni sosoliso ko dana ni ... Aint gone waste my bars on you. ![]() |
JigsawKillah: how can someone buy a 'under'?Under- Honda. . .Your dumb as fucc. Ion quote my puns, w/p like you do. If your d0pe, you gone get em... Bytch! I'll reply your wack verse later. Bold |
missyhorlah: Yesterday I was passing by a supermarket!so I dcided to go in nd pick a drink and a gala!as I walked in!I noticed a man was staring at me,so I returned a fake smile and then ad a walk down d aisle to pick my can malt....only to notice that the man was still ffg me!I was kinda getting scared buht I tried to be polite nd I said good afternoon sir!d man didn't reply me so I proceeded !then d man tapped me frm behind nd said m sorry af bin staring at u for a while now,buht u af a similar features wv my dead dota!I was like wtfyou a fcking Liar... I seen this a million times |
mad men for your side dey chop refuse ... Do they live long . .wtf! Too much of Nigerian films... |
JigsawKillah: read dis with massive-attention, ur acting-with-tension, just like ur dad always bragging-on-pension, ur monoflow is as dumb as naija movies where u see a ghost ask-for-direction'sleeptyping, bytch! I'll come with a lighter to burn down your roof, your dad be saying- He's smoking hot/ Getit?? You guys live in a hut, your broke as fucc/. . And last week my dad bought a Range Rover sport/. . . When your dad couldn't afford a under, you keep lying he flys over us/ Bytch! The dude your mentor gets inspirations from is whom I'm Ghosting for/. . You a bloody fagg, you damn choke on coccs/. . Once he sees a guy with fat buttt, Dayum! He gets soaked up like Golden morn/. . You beefing mr? Your dead, pauper!/ I'm a cooger, you a cat! You should dread, brother!/. . You run the game?? 'Shoot you right on track then I call that step murder!!! Bytch!! Must you drag your PS1 on the stairs to step up your game??/ stop dragging, kid! You lacking it. . .Your verses are lame/. . . Cos like I'm a student 'bout to finish a course, fagg! I'm ending your fate!!! |
JigsawKillah: ur life's a movie, M-B-C-2, u must be an as5, the way im messing-with-u, ur faker than a hologram, i'll let u drown in a flood of flames, hollowmanSLOW POISON Elvis knows he a sore looser/. . . Now he created a new moniker/ You wanna troll? I dropped that when you P.man stopped wearing short knickers/. . . You a puCC on menses- a bloody cat/. . . Ion need to punch his buttt. . . to knock his asss/. . . .outta here Dumbfuck!. . .married to a bossy dam/ his best food his maize, now he thinks he's a con Mister/. . Wtf!!! Funny fagg/. . . He gets beaten in his own noisy park/ Then, he goes to practise. . .always blowing the trees- saying hes coming back/ Bytch! My punchlines punch you and what your punchlines do . .They just sit back/. . .Cos you always popping dummy bars/ spitting d0pe bars aint notting hard/. . Dummy!!! You should bite bars like a dog in a cage. . . or meow lines be a phuckin cat!!! Po! Po!! Po!!!/ 'just killed a bytch with Monoflow. . . |


